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- W2185402912 abstract "using CCOantibody. Thesefindings suggest that partical CCO deficiency maybepresent intheskeletal muscles ofthepatient. NADH cytochrome creductase inthepatient's muscle mitochondria waslowcompared withnormal controls (about 26%),although succinate cytochrome creductase wasnormal. Coenzyme Q1O administration (90mg/day) didnotimprove CSFlactate levels, butdiddecrease plasma lactate levels. Hismuscle weakness slightly improved. Mitochondrial myopathy denotes agroupofvarious disorders withmitochondrial abnormalities. In1977, Shapira etal' noted agroup ofdiseases characterised bycentral nervous system (CNS)symptoms, and advocated thetermmitochondrial encephalomnyopathy, suggesting thepresence ofa common metabolic disorder inthemuscle andCNS.Among patients withthis disorder, Pavlakis etal2described twooftheir ownandnineintheliterature showing mitochondrial myopathy, encephalopathy, lactic acidosis, andstrokelike episodes (MELAS), andconsidered this condition tobeadistinct clinical entity. We examined enzymeactivity inthemitochondrial electron transport system inapatient withMELAS, andstudied theeffects oftheadministration of coenzyme Q0 (CoQO0), animportant component ofthesystem. Clinical features aswell astheresults of th-istreatment arereported." @default.
- W2185402912 created "2016-06-24" @default.
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- W2185402912 date "1987-01-01" @default.
- W2185402912 modified "2023-09-24" @default.
- W2185402912 title "Mitochondrial myopathy, encephalopathy, lactic acidosis, andstrokelike episodes withrecurrent abdominal symptomsandcoenzyme Q10 administration" @default.
- W2185402912 cites W1819875704 @default.
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