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- W2185830695 abstract "was present atthe inactive one.We suggest that centromere inactivation results froma modified conformation of thefunctional DNA sequencespreventing normal binding tocentromere specific proteins. We also postulate that thepatient's disorder, reminiscent ofa spongyglioneuronal dystrophy asseen inAlper's andCreutzfeldt-Jakob diseases, may besecondary tothepresenceofthepathogenic isoform oftheprion protein encoded bya genemappedto20p12-3-pter." @default.
- W2185830695 created "2016-06-24" @default.
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- W2185830695 date "1989-01-01" @default.
- W2185830695 modified "2023-09-27" @default.
- W2185830695 title "Alternate centromere inactivation inapseudodicentric" @default.
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- W2185830695 hasPublicationYear "1989" @default.
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