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• W2185879107 abstract "Summary: lesions, and magnetic resonance imaging of the bone marrow was normal. Electrocardiogram and echocardiography AL amyloidosis is an infiltrative disorder characterized were normal. by the extracellular deposition of insoluble fibrillar The patient had an HLA-identical sister. As first-line immunoglobulin light chains whose production results therapy, an allogeneic BMT was performed on 24 August from a plasma cell dyscrasia. Treatment with melphalan 1995. Conditioning consisted of melphalan, 140 mg/m 2 , has resulted in an improvement in a few patients. and TBI (800 cGy, single dose). GVHD prophylaxis Recently, intensive chemotherapy followed by autolog- included CsA and MTX. The immediate post-grafting perous or syngeneic stem cell support has been shown to iod was uneventful. Grade I GVHD progressing to grade II offer potential benefit. Allogeneic stem cell support after developed on day 30 post-transplant. Methylprednisolone, intensive therapy would retain the benefits of autolog- 2 mg/kg/day for 10 days, followed by tapering of the dose, ous transplantation, with the additional advantages of was effective. The monoclonal component was no longer a tumor-free graft and of a possible graft-versus-tumor detected in blood and urine on 25 October 1995. On effect. We report a patient with AL amyloidosis and sig- 17 November 1995 the bone marrow was normal, without nificant proteinuria. She improved after an allogeneic abnormal plasma cells and without B cell monoclonality on bone marrow transplantation. Southern blot analysis. There was full chimerism as demon" @default.
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• W2185879107 date "1997-01-01" @default.
• W2185879107 modified "2023-09-26" @default.
• W2185879107 title "Case report Allogeneic bone marrow transplantation for AL amyloidosis" @default.
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