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- W2186919102 abstract "his paper examines speech pathologies, specifically those of three aphasic patients, and diagnoses their form of aphasia by analysing transcripts of their speech. The approach I will follow is to consider the principle characteristics of three different forms of aphasia, and by analysing the patients oral deficits and difficulties decide which type of aphasia most resembles the symptoms they display. The three aphasias I will be focusing on are Wernicke‟s, Broca‟s and anomic aphasia. Elizabeth Ahlsen defines aphasia as “a language disorder caused by acquired brain damage” (2006: 101), and the effect of the brain lesion is that “comprehension and/or production of language is changed” (2006: 6). There appears to be no agreed number of forms of aphasia, though they tend to be divided into two groups: fluent and nonfluent aphasics. Different views as to how the brain processes language have led to various theories as to what determines the type of aphasia. For example, the holistic approach, supported by Pierre Marie, amongst others, proposes that the brain functions as a single unit, and there is a single basic form of aphasia: sensory aphasia (Triarhou, Tsapkini and Vivas, 2008). From an holistic perspective aphasia is part of a general cognitive loss rather than a specific language deficit related to a particular area of the brain (Ahlsen, 2006). This bears a similarity to the idea of equipotentiaility, which suggests all parts of the cortex are capable of the same functions and that “the size of the brain lesion determines the extent of the aphasia” (Ahlsen, 2006: 11), rather than taking into account where the lesion is. This contrasts with the localistassociasionist model, a theory supported more recently by Norman Geschwind (Farah and Feinberg, 2000), in which different parts of the brain are responsible for different aspects of language comprehension and processing; therefore the site is of the lesion is important, as well as its size, in determining the type of aphasia. Wernicke‟s aphasia generally results from posterior lesions in the Wernicke‟s region of the left hemisphere. Kent suggests that Wernicke‟s aphasia “affects 15%-25% of all patients with aphasia” (2003: 252) and that it is most prevalent amongst older patients (Kent, 2003). It is a fluent form of aphasia with longer utterances of six words or more (Kent, 2003), though often lacking meaning. Friedemann Pulvermuller states that the main symptoms of Wernicke‟s aphasia are that patients “use words in inappropriate contexts; produce words incorrectly, with incorrect language sounds in them or language sounds omitted...” (2003: 36). Ahlsen has suggested that Wernicke‟s patients also omit certain kinds of words “with a relative lack of nouns in particular, but also adjectives and main verbs and with substitutions of grammatical morphemes” (2006: 68), which makes it harder to discern the meaning. Kent (2003) amongst others categorises Wernicke‟s aphasia as aggrammatic. However, this is contested by neurolinguists such as Kaplan (McCaffrey, 1998-2008), who suggests that Wernicke‟s patients are paragrammatic and produce the wrong grammatical categories but which are relatively syntactically complex in themselves. Others have suggested that it is not possible to distinguish between agrammatic and paragrammatic speakers from spontaneous speech and that patients may display traits of both (Miceli, 1999). T Volume 1: 2008-2009 ISSN: 2041-6776" @default.
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- W2186919102 date "2009-01-01" @default.
- W2186919102 modified "2023-09-27" @default.
- W2186919102 title "Language and Development: Diagnosing Speech Pathologies" @default.
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