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- W2188107608 abstract "Rheumatic fever was first described in the nineteenth century. Though potentially preventable, it continues to be a major health problem and is still a major cause of mortality from heart disease during the first 50 years of life. The incidence of rheumatic fever in the western world began to decline long before the introduction of antibiotic treatment. In Denmark, for example, the incidence of the disease decreased from 250 per 100,000 population in 1862 to 100 per 100,000 in 1962 [1]. This trend was rapidly accelerated with the introduction of antibiotics, and the incidence reported in 1980 was less than 2 per 100,000. In developing countries, however, the incidence is still high and is up to 13.4 per 100,000 hospitalized children [2]. According to current estimates, 10–20 million new cases per year are expected in these countries [3]. Nevertheless, the sudden reappearance of focal epidemics in the United States in the mid-1980s reminded us that the disease still poses a threat worldwide [4]. A better understanding of the pathogenesis of RF is needed to explain its changing incidence. Despite the expanding epidemiological and immunological evidence linking group A Streptococcus infection to RF, many unanswered questions remain. Studies have shown that not all group A streptococcal strains are rheumatogenic. It is known that pyoderma and soft tissue infections never cause rheumatic fever. Furthermore, even though the pharynx is the site of the antecedent rheumatic infection, not all group A streptococcal pharyngeal infections lead to RF. Though opinions vary regarding the rheumatogenicity of certain streptococcal strains [3], several factors do support their existence, namely:" @default.
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- W2188107608 date "2000-06-01" @default.
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- W2188107608 title "Rheumatic fever: a never-ending story?" @default.
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