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- W2189724894 startingPage "239" @default.
- W2189724894 abstract "Endothelial dysfunction (ED), an early indicator of vascular disease, is common in type 2 diabetes and independently predicts cardiovascular disease. The pathogenesis of ED in type 2 diabetes is complex. Although the precise pathogenetic mechanisms for ED remain unclear, at inception they probably involve uncoupling of both eNOS activity and mitochondrial oxidative phosphorylation, as well as the activation of vascular NAD(P)H oxidase. The major contributing factors include dyslipoproteinaemia, oxidative stress and inflammation. Therapeutic interventions, including lifestyle changes and lipid-regulating therapies, correct diabetic dyslipidaemia and can improve ED. High residual cardiovascular risk is seen with several interventions in both research and clinical practice settings. Well designed studies of endothelial function in appropriately selected volunteers afford a good opportunity to test new therapeutic interventions, paving the way for clinical trials and utilization in the care of the diabetic patient." @default.
- W2189724894 created "2016-06-24" @default.
- W2189724894 creator A5008958109 @default.
- W2189724894 creator A5070816464 @default.
- W2189724894 date "2013-07-17" @default.
- W2189724894 modified "2023-09-28" @default.
- W2189724894 title "Endothelial Dysfunction and Dyslipidemia in Type 2 Diabetes: Pathogenesis, Significance and Therapy" @default.
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