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• W2193312241 endingPage "625" @default.
• W2193312241 startingPage "615" @default.
• W2193312241 abstract "Air pollution is a major problem faced globally and is seen associated with central nervous system (CNS) disorders like neuropathology and neuro-inflammation. Here, we investigated the CNS disorders as a result of sub-chronic exposure (90 days) to diesel exhaust nanoparticles (DENPs) and explored the minimal levels of DENPs needed to exhibit the early mediators of neuro-inflammation and neuropathology. Male and female wistar rats (6 rats per group) were exposed to DENPs (1/5th, 1/10th and 1/15th LC50) by inhalation for 4h per day, 5 days per week over 90 days and neurotoxicity end-points were analyzed. DENP exposure caused elevation in levels of pro-inflammatory cytokines, amyloid beta 42 (Aβ 42), reactive oxygen species (ROS), hydrogen peroxide (H2O2), nitrate (NO3(-)), nitrite (NO2(-)) and apurinic/apyrimidinic sites (AP) at varying degrees at different sections of rat brain. Hence, exposure to DENPs resulted in dose-dependent toxicity and was closely correlated to increased inflammation, DNA damage and oxidative stress." @default.
• W2193312241 created "2016-06-24" @default.
• W2193312241 creator A5030030484 @default.
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• W2193312241 date "2015-09-01" @default.
• W2193312241 modified "2023-10-01" @default.
• W2193312241 title "Determination of LC50 and sub-chronic neurotoxicity of diesel exhaust nanoparticles" @default.
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• W2193312241 doi "https://doi.org/10.1016/j.etap.2015.06.024" @default.
• W2193312241 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/26363131" @default.
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