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- W2196811906 abstract "Both Alzheimer’s disease (AD) and Parkinson’s disease (PD) exist in rare familial forms caused by mutations in genes that encode the predominant fibrillar protein component of the characteristic lesion: amyloid plaques and Lewy bodies, respectively. This convergence of pathological and genetic information has given rise to the idea that the fibrils themselves are responsible for neuronal death. Biophysical studies, summarized herein, have demonstrated that the mutations accelerate the overall process of fibril formation, including the accumulation of an intermediate species, designated the protofibril. We propose that protofibrils, rather than fibrils, are the pathogenic entities in both AD and PD." @default.
- W2196811906 created "2016-06-24" @default.
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- W2196811906 date "2000-01-01" @default.
- W2196811906 modified "2023-09-27" @default.
- W2196811906 title "The Role of α-Synuclein in Parkinson’s Disease: A Biophysical Analogy to Aβ and Alzheimer’s Disease" @default.
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- W2196811906 doi "https://doi.org/10.1007/978-3-662-04056-0_1" @default.
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