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• W2199628131 endingPage "15712" @default.
• W2199628131 startingPage "15707" @default.
• W2199628131 abstract "Significance Circadian rhythms are common to most eukaryotes, and, in humans, circadian dysfunction is associated with sleep disorders, elevated incidence of cancer, and metabolic abnormalities. The biological oscillators giving rise to fungal and animal clocks share a common regulatory architecture, as well as some highly conserved components, many of which were identified in genetic screens. Here a gene, period -1 ( prd -1), previously identified in a genetic screen in the filamentous fungus Neurospora , is shown to encode a protein (an ATP-dependent RNA helicase) that is closely related to yeast and human proteins involved in RNA metabolism and transcription. The cycle length in prd -1 mutants is only abnormal when available glucose is elevated, suggesting that normal clock regulation in response to nutrition is altered." @default.
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• W2199628131 date "2015-12-08" @default.
• W2199628131 modified "2023-09-26" @default.
• W2199628131 title "<i>period</i> -1 encodes an ATP-dependent RNA helicase that influences nutritional compensation of the <i>Neurospora</i> circadian clock" @default.
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• W2199628131 doi "https://doi.org/10.1073/pnas.1521918112" @default.
• W2199628131 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/4697410" @default.
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