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- W2201212080 abstract "Despite several decades of work there is still no evidence proving that tissue kallikrein and its kinin products have any causal, or compensatory role in any form of experimental or human hypertension. Nevertheless, such participation continues to be reasonable because of findings which established that: 1) changes in endogenous renal kallikrein-kinin activity are correlated with changes in renal excretory or hemodynamic behavior (1,2); 2) correlations are present between urinary kallikrein excretion rates and levels of blood pressure in some normal children (3); 3) altered urinary and renal kallikrein levels are present in various forms of renal and hypertensive diseases (4,5); and 4) changes in system component activity occur during treatment with antihypertensive and/or diuretic drugs (6)." @default.
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- W2201212080 date "1984-01-01" @default.
- W2201212080 modified "2023-09-27" @default.
- W2201212080 title "Kallikrein and Kinins in Epithelial Ion Transport" @default.
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- W2201212080 doi "https://doi.org/10.1007/978-1-4613-3897-0_5" @default.
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