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• W2202241795 abstract "Significance Eukaryotes and bacteria are often in dialogue, in some cases mutualistic and in other cases pathogenic. A highly evolved system of measures and countermeasures by both bacteria and eukaryotic host may mediate these interactions. Here we found that mutations in the electron transport pathway of the normally benign Escherichia coli , which is a food for Caenorhabditis elegans in the laboratory, induces a mitochondrial stress response. The oxidatively stressed E. coli activates mitochondrial stress responses via transcription factors that are known to mediate mitochondrial homeostasis. Activation of C. elegans mitochondrial responses could be suppressed by additional mutations in E. coli response pathways for free radical damage, showing that the animal response depends on bacterial response to free radical damage." @default.
• W2202241795 created "2016-06-24" @default.
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• W2202241795 date "2015-09-21" @default.
• W2202241795 modified "2023-10-16" @default.
• W2202241795 title "Dialogue between <i>E. coli</i> free radical pathways and the mitochondria of <i>C. elegans</i>" @default.
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• W2202241795 doi "https://doi.org/10.1073/pnas.1517448112" @default.
• W2202241795 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/4603447" @default.
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