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- W2202777379 abstract "Radiation therapy is frequently used in the treatment of a number of different tumors. However, the effectiveness of radiotherapy is limited by the ability of normal tissues adjacent to tumors to tolerate radiation in the doses required to kill or sterilize tumor cells. This limitation is compounded by the presence in tumors of radiation-resistant cells that may arise as a result of environmental factors, such as hypoxic regions in tumors, the expression of growth factors that can reduce radiation sensitivity, or tumor cell intrinsic radiation resistance that may be imparted through the activation of certain oncogenes. Ras oncogenes in particular may contribute to radiation resistance, because they have been shown to increase radiation resistance in many experimental systems, and are mutated in an estimated 30% of all human tumors. Basic fibroblast growth factor (bFGF) has also been implicated in increased radiation resistance and is over-expressed in certain tumors, particularly glioblastomas." @default.
- W2202777379 created "2016-06-24" @default.
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- W2202777379 date "2001-01-01" @default.
- W2202777379 modified "2023-09-23" @default.
- W2202777379 title "Prenyltransferase Inhibitors as Radiosensitizers" @default.
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- W2202777379 doi "https://doi.org/10.1007/978-1-59259-013-1_12" @default.
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