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- W2209545137 abstract "Abstract The human fetus is an active participant in its own development by integrating information received from its maternal host into its neurodevelopmental program to prepare for life after birth. Because the fetal nervous system develops at precise times and in a specific sequence from conception to birth, disruption by intrauterine events in the timing or sequence of brain development results in tissue remodeling. Depending on the timing of the disruption, the function and physiological capacity of the remodeled brain is affected throughout the lifespan. Evidence from prospective and retrospective studies supports the conclusion that fetal exposures to maternal signals of psychobiological stress or being born small or early are associated with increased risk for behavioral disorders and alterations in brain structures in children and adults. A range of early-life exposures results in similar neurobehavioral “programs” suggesting a common neural mechanism. Alteration in dendritic tree complexity is one plausible mechanism that mediates the relation between fetal exposure to stress and neurobehavioral programming." @default.
- W2209545137 created "2016-06-24" @default.
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- W2209545137 date "2016-01-01" @default.
- W2209545137 modified "2023-09-26" @default.
- W2209545137 title "Neurobehavioral Disorders and Developmental Origins of Health and Disease" @default.
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