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• W2209807187 abstract "Pseudo-Cushing’s syndromes (PCS) are defined as states associated with increased cortisol production with all or some of the clinical features of Cushing’s syndrome (CS), combined with biochemical evidence of hypercortisolism, but without evidence for a neoplastic etiology. These states include conditions such as severe physical or emotional stress, alcoholism or alcohol withdrawal, and chronic medical and psychiatric diseases such as depression and various psychoses. Although resolution of the primary condition usually leads to disappearance of the Cushingoid features and hypercortisolism, the underlying mechanisms remain unclear. Most evidence suggests that central stimulation of corticotropin-releasing hormone (CRH), either at the hypothalamic or supra-hypothalamic level, is responsible. This leads to characteristic alterations in endocrine testing aimed at differentiating PCS from true states of excessive cortisol production. Distinguishing PCS from CS is clinically relevant, because a patient with a PCS who is misdiagnosed as CS may undergo needless testing and potentially harmful treatments, while missing the diagnosis of CS can lead to increased morbidity and mortality risk from the underlying disease." @default.
• W2209807187 created "2016-06-24" @default.
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• W2209807187 date "2011-01-01" @default.
• W2209807187 modified "2023-09-26" @default.
• W2209807187 title "Pseudo-Cushing’s Syndrome" @default.
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• W2209807187 doi "https://doi.org/10.1007/978-1-4614-0011-0_5" @default.
• W2209807187 hasPublicationYear "2011" @default.
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