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- W2215601219 abstract "Acute hypotensive blood loss leads to increased ventilation as evidenced by tachypnea, hyperoxia, and hypocapnia (FASEB J. 18:A1277, 2004). Since blood loss is normally accompanied by stress, we investigated the respiratory response to HH in the presence of a stressful stimulus (AIR). We hypothesized that AIR would alter the respiratory and blood gas changes induced by HH. The cardiovascular and respiratory response to hemorrhage in the presence of sham stress (SHAM) and AIR were compared in 10 rabbits chronically instrumented with arterial and venous catheters and diaphragmatic EMG electrodes. Heart rate (HR), mean arterial pressure (MAP), respiratory rate (RR), and arterial blood gases were measured before hemorrhage, after normotensive hemorrhage, and after HH (MAP 40 mmHg). Before hemorrhage, AIR increased HR, MAP, and RR, and decreased PaCO2 [213±6 bpm (mean ± SE), 91±2 mmHg, 246±15 bpm, and 35±1 mmHg, respectively] compared to SHAM (159±5 bpm, 73±2 mmHg, 121±17 bpm, and 37±1 mmHg, respectively). These differences were maintained through normotensive hemorrhage. After HH, HR, RR, and PaCO2 were significantly different between AIR (282±16, 265±16, and 27±1, respectively) and SHAM (159±5, 130±15, and 30±1, respectively). PaO2 increased significantly at each timepoint, but was not different between AIR and SHAM. These results suggest that AIR induces tachypnea and hypocapnia that is maintained throughout HH. In addition, despite the relative hyperventilation caused by AIR, the ventilatory response to HH was unaltered. Supported by NIH and ONR." @default.
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- W2215601219 date "2006-03-01" @default.
- W2215601219 modified "2023-09-26" @default.
- W2215601219 title "Respiratory response to hypotensive hemorrhage (HH) in the presence of air jet stress (AIR)" @default.
- W2215601219 doi "https://doi.org/10.1096/fasebj.20.4.a376-a" @default.
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