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- W2220872129 abstract "While there is general agreement that angina pectoris results from a deficit of myocardial blood flow relative to the myocardial metabolic demands [10], the approaches to ameliorate this imbalance by drugs have changed remarkably within the last decades. Drugs increasing coronary flow in the normal heart by coronary arteriolar dilation have been abandoned because of the risk that they might induce “coronary steal” in a heart with diseased coronary arteries [3]. Subsequently, extra-cardiac actions of antianginal drugs (balanced reduction of cardiac preload and afterload), indirect improvement of coronary flow (by lowered demand and extra-vascular coronary resistance), and blockade of sympathetic augmentation of myocardial contractility were regarded as important for therapy." @default.
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- W2220872129 date "1985-01-01" @default.
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- W2220872129 title "Effect of Different Vasodilators on Epicardial Coronary Arteries" @default.
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