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- W2222077151 endingPage "104" @default.
- W2222077151 startingPage "95" @default.
- W2222077151 abstract "GPCRs are ubiquitous in mammalian cells and present intricate mechanisms for cellular signaling and communication. Mechanistically, GPCR signaling was identified to occur vectorially through heterotrimeric G proteins that are negatively regulated by GRK and arrestin effectors. Emerging evidence highlights additional roles for GRK and Arrestin partners, and establishes the existence of interconnected feedback pathways that collectively define GPCR signaling. GPCRs influence cellular dynamics and can mediate pathologic development, such as cancer and cardiovascular remolding. Hence, a better understanding of their overall signal regulation is of great translational interest and research continues to exploit the pharmacologic potential for modulating their activity." @default.
- W2222077151 created "2016-06-24" @default.
- W2222077151 creator A5020601372 @default.
- W2222077151 creator A5028159080 @default.
- W2222077151 creator A5050538530 @default.
- W2222077151 date "2016-02-01" @default.
- W2222077151 modified "2023-10-12" @default.
- W2222077151 title "Feedback regulation of G protein-coupled receptor signaling by GRKs and arrestins" @default.
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