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• W2224901024 abstract "Exon 19 deletions and point mutations in L858R are the most common somatic activating mutations in the epidermal growth factor receptor (EGFR) gene that confer sensitivity to EGFR tyrosine kinase inhibitors (TKIs) in lung cancer.1 However, despite the initial response to EGFR TKIs, all patients will eventually develop resistance. One of the most common mechanisms of resistance is acquisition of a second mutation at exon 20, which causes a T790M substitution.2,3 Although most of these cases have acquired resistance through somatic mutations, a small number of germline EGFR T790M have been reported and are estimated to occur in 1% of non–small-cell lung cancer (NSCLC) cases." @default.
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• W2224901024 date "2016-03-01" @default.
• W2224901024 modified "2023-10-18" @default.
• W2224901024 title "Germline Mutation of T790M and Dual/Multiple EGFR Mutations in Patients With Lung Adenocarcinoma" @default.
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• W2224901024 doi "https://doi.org/10.1016/j.cllc.2015.11.003" @default.
• W2224901024 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/5119523" @default.
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