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- W2227022280 abstract "For the last two decades there has been an enormous effort to find therapeutic means of protecting the ischemic and reperfused myocardium. Numerous studies concentrated on finding and targeting specific pharmacological agents in an attempt to reduce experimental infarct size, with the ultimate clinical aim of limiting postinfarct morbidity and mortality [1,2]. Unfortunately such agents never came to the fore, and it was accepted that drugs may at best have the ability to delay ischemia/reperfusion injury without an appreciable effect on ultimate myocardial infarct size [3,4]." @default.
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- W2227022280 date "1994-01-01" @default.
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- W2227022280 title "Stress Proteins, Heat Stress, and Myocardial Protection" @default.
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