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• W2234309697 abstract "This chapter highlights recent progress made toward understanding the molecular basis of Salmonella-induced enteritis. Information is scarce on the specific course of events following nontyphoidal Salmonella infection in humans because most infected individuals are rarely hospitalized. As such, observations of nontyphoidal Salmonella infection have mostly come from patients admitted to the hospital with severe fatal infections. Interactions between bacteria and intestinal tissue were examined in starved, opium-treated guinea pigs several hours after oral challenge with 108 invasive serovar Typhimurium. This study found that Salmonella closely contacts the epithelial cells lining the intestine, primarily the ileum, and thereafter elicits the local degeneration of filamentous actin in apical microvilli and the underlying terminal web. More recently, studies have turned to the use of cattle to model the pathophysiology of Salmonella-induced enteritis in humans. Following nitrogen mustard administration, the rabbit ligated ileal loops were then infected with serovar Typhimurium for 72 h. Results from this study revealed that nitrogen mustard treatment markedly inhibited serovar Typhimurium-induced secretion. This inflammatory response greatly contributes to the pathophysiology of the infection, exhibited by typical inflammatory diarrhea. Recent work has begun to disclose the molecular and cellular events involved in this complex phenomenon. In addition to TTSS-1-associated genes, several other genes affecting Salmonella enteropathogenicity are currently being investigated." @default.
• W2234309697 created "2016-06-24" @default.
• W2234309697 creator A5068098793 @default.
• W2234309697 date "2014-04-09" @default.
• W2234309697 modified "2023-09-26" @default.
• W2234309697 title "<i>Salmonella</i>spp.: Masters of Inflammation" @default.
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• W2234309697 doi "https://doi.org/10.1128/9781555817848.ch24" @default.
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