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- W2235138889 abstract "While the basic principles of radiation chemistry and radiation molecular biology have not changed, there has been significant modification in our understanding of cellular interactions involved in irradiation tissue damage [1, 2, 3, 4, 5]. Ionizing irradiation induces radiation chemical changes in cells, principally targeting oxygen and water molecules which results in formation of superoxide, hydroxyl, and other free radical moieties, within fractions of a second after radiation exposure [2]. Formation of nitric oxide, and its combination with superoxide leads to formation of peroxynitrite, a potent pro-oxidant [6, 7], which in combination with the other radical oxygen species leads to significant lipid peroxidation as well as direct binding to nuclear DNA that results in single and double strand breaks [8, 9, 10, 11]. In the last decade, it has become clear that DNA strand breaks are repaired rapidly, certainly within 15 min after irradiation of cells in culture [4, 5], and that this repair involves a complex interaction of multiple rapid response genes, which initiate by site specific phosphorylation of the ataxia telangiectasia protein (ATM) on a specific phosphorylation site [12]." @default.
- W2235138889 created "2016-06-24" @default.
- W2235138889 creator A5063266916 @default.
- W2235138889 date "2008-01-01" @default.
- W2235138889 modified "2023-09-27" @default.
- W2235138889 title "Bioengineering in the Repair of Irradiated Normal Tissue by Bone Marrow Derived Stem Cell Populations" @default.
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