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• W2235539938 abstract "To investigate the role of the non-receptor tyrosine kinase c-Abl in high glucose-induced podocyte injury and its possible signal transduction pathway.Sixteen C57BL/6 mice were randomly assigned to a group with diabetes and a normal control group. Subsequently, differentiated mouse podocytes were exposed to high-glucose conditions, and podocyte apoptosis was then assessed by flow cytometry and Hoechst 33258 staining. Western blot and immunofluorescence assay were used to measure c-Abl expression. Co-immunoprecipitation assay was used and c-Abl siRNA was applied to evaluate the interaction between c-Abl and p53.High glucose promotes podocyte apoptosis. The c-Abl expression in podocytes was increased after exposure to high glucose, stimulating the p53 signaling pathway. Conversely, treatment with c-Abl siRNA restored high glucose-promoted podocyte apoptosis and resulted in the reduction of p53 expression.c-Abl contributes to high glucose-induced podocyte apoptosis via p53 signaling pathway." @default.
• W2235539938 created "2016-06-24" @default.
• W2235539938 creator A5003281764 @default.
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• W2235539938 date "2016-03-01" @default.
• W2235539938 modified "2023-10-01" @default.
• W2235539938 title "c-Abl contributes to glucose-promoted apoptosis via p53 signaling pathway in podocytes" @default.
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• W2235539938 doi "https://doi.org/10.1016/j.diabres.2015.12.013" @default.
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