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• W2236379301 startingPage "114" @default.
• W2236379301 abstract "Chronic inflammation, imbalance between the extracellular matrix synthesis and degradation, and loss of vascular smooth muscle cells (SMCs) contribute to the development of abdominal aortic aneurysm (AAA). The purpose of this study was to investigate the effect of the therapy with periaortic incubation of c-Jun N-terminal protein kinase inhibitor SP600125 infused from an osmotic pump and subadventitial injection of lysyl oxidase (LOX) gene modified autologous smooth muscle progenitor cells (SPCs) on treatment of AAA in a rabbit model. Obvious dilation of the abdominal aorta in the control group was caused by periaortic incubation of calcium chloride and elastase. But the progression of aortic dilation was significantly decreased after the treatment with SP600125 and LOX gene modified SPCs compared to the treatment with phosphate-buffered saline. This therapy could inhibit matrix metalloproteinases expression, enhance elastin synthesis, improve preservation of elastic laminar integrity, benefit SPCs survival and restore SMCs population. It seemed that this method might provide a novel therapeutic strategy to treat AAA." @default.
• W2236379301 created "2016-06-24" @default.
• W2236379301 creator A5060808721 @default.
• W2236379301 creator A5080815812 @default.
• W2236379301 creator A5081609090 @default.
• W2236379301 date "2015-11-01" @default.
• W2236379301 modified "2023-09-27" @default.
• W2236379301 title "Inhibition of development of experimental abdominal aortic aneurysm by c-jun N-terminal protein kinase inhibitor combined with lysyl oxidase gene modified smooth muscle progenitor cells" @default.
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• W2236379301 doi "https://doi.org/10.1016/j.ejphar.2015.09.046" @default.
• W2236379301 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/26435026" @default.
• W2236379301 hasPublicationYear "2015" @default.
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