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• W2242204003 abstract "Deletion of the beta3 subunit of the GABA(A) receptor produces severe behavioral deficits and epilepsy. GABA(A) receptor-mediated miniature inhibitory postsynaptic currents (mIPSCs) in cortical neurons in cultures from beta3 -/- mice were significantly faster than those in beta3 +/+ mice and were more prolonged by zolpidem. Surface staining revealed that the number of beta2/3, alpha2, and alpha3 (but not of alpha1) subunit-expressing neurons and the intensity of subunit clusters were significantly reduced in beta3 -/- mice. Transfection of beta3 -/- neurons with beta3 cDNA restored beta2/3, alpha2, and alpha3 subunits immunostaining and slowed mIPSCs decay. We show that the deletion of the beta3 subunit causes the loss of a subset of GABA(A) receptors with alpha2 and alpha3 subunits while leaving a receptor population containing predominantly alpha1 subunit with fast spontaneous IPSC decay and increased zolpidem sensitivity." @default.
• W2242204003 created "2016-06-24" @default.
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• W2242204003 date "2003-01-01" @default.
• W2242204003 modified "2023-10-13" @default.
• W2242204003 title "GABA_AReceptor β3 Subunit Deletion Decreases α2/3 Subunits and IPSC Duration" @default.
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• W2242204003 doi "https://doi.org/10.1152/jn.00700.2002" @default.
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