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- W2255673114 endingPage "903" @default.
- W2255673114 startingPage "893" @default.
- W2255673114 abstract "The neurovascular unit, which consists of astrocytic end-feet, neurons, pericytes, and endothelial cells, plays a key role in maintaining brain homeostasis by forming the blood-brain barrier and carefully controlling local cerebral blood flow. When the blood-brain barrier is disrupted, blood components can leak into the brain, damage the surrounding tissue and lead to cognitive impairment. This disruption in the blood-brain barrier and subsequent impairment in cognition are common after stroke and during cerebral amyloid angiopathy and Alzheimer's disease. Matrix metalloproteinases are proteases that degrade the extracellular matrix as well as tight junctions between endothelial cells and have been implicated in blood-brain barrier breakdown in neurodegenerative diseases. This review will focus on the roles of MMP2 and MMP9 in dementia, primarily post-stroke events that lead to dementia, cerebral amyloid angiopathy, and Alzheimer's disease." @default.
- W2255673114 created "2016-06-24" @default.
- W2255673114 creator A5005215398 @default.
- W2255673114 creator A5010321411 @default.
- W2255673114 date "2016-01-01" @default.
- W2255673114 modified "2023-10-16" @default.
- W2255673114 title "Matrix Metalloproteinase in Blood-Brain Barrier Breakdown in Dementia" @default.
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