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- W2257800414 endingPage "275" @default.
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- W2257800414 abstract "Histologic bone changes of osteitis fibrosa and osteomalacia are commonly present in patients with end-stage renal disease. Although many patients are not symptomatic from these bone changes, some patients are severely disabled. Altered metabolism of vitamin D, calcium, phosphorus, and parathyroid hormone occurs in renal failure and contributes to the development of uremic bone disease. This article reviews the current theories of pathogenesis and treatment of renal osteodystrophy. In addition, the clinical presentation, pathogenesis, and treatment of the various aluminum-associated osteomalacic syndromes in uremia are discussed." @default.
- W2257800414 created "2016-06-24" @default.
- W2257800414 creator A5024128744 @default.
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- W2257800414 date "1986-04-01" @default.
- W2257800414 modified "2023-09-26" @default.
- W2257800414 title "Review: Renal Osteodystrophy—Pathogenesis Treatment" @default.
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- W2257800414 doi "https://doi.org/10.1097/00000441-198604000-00008" @default.
- W2257800414 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/3518448" @default.
- W2257800414 hasPublicationYear "1986" @default.
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