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- W2257876566 endingPage "190" @default.
- W2257876566 startingPage "177" @default.
- W2257876566 abstract "Accumulating evidence suggests a role for inflammation in the pathophysiology of epilepsy. We performed a systematic review and meta-analysis of studies that investigated inflammatory mediators in human epilepsy. Studies reporting on inflammatory mediators in serum, cerebrospinal fluid or brain tissue of epilepsy patients were included. Studies comparing patients to controls were included in a meta-analysis. 66 articles reporting on 1934 patients were included. IL-1ra, IL-1β, IL-6, IL-10, IFN-γ and TNF-α were the most extensively investigated proteins. Elevated levels for IL-1ra, IL-1β, IL-6 and CXCL8/IL-8 were reported in several different epilepsy etiologies and media, while other proteins were specifically increased for one etiology. IL-1α, IL-7 and IL-13, as well as the chemokines CCL2-5, −19 and −22, were increased exclusively in brain tissue. In an aggregate meta-analysis, we found significantly different protein levels for serum IL-6, IL-17 and CSF IL-1β and IL-10. Inflammatory pathways are involved in epilepsy. Future studies may further clarify their role, and prove potential of targeted anti-inflammatory treatment." @default.
- W2257876566 created "2016-06-24" @default.
- W2257876566 creator A5003603164 @default.
- W2257876566 creator A5039140743 @default.
- W2257876566 creator A5041084071 @default.
- W2257876566 creator A5055575694 @default.
- W2257876566 creator A5056152125 @default.
- W2257876566 creator A5064705806 @default.
- W2257876566 date "2016-04-01" @default.
- W2257876566 modified "2023-09-26" @default.
- W2257876566 title "Inflammatory mediators in human epilepsy: A systematic review and meta-analysis" @default.
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