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- W2258124546 endingPage "306" @default.
- W2258124546 startingPage "281" @default.
- W2258124546 abstract "Enhancement of reactive oxygen species (ROS) production impairs the physiological function of the cardiovascular system due to activation of specific signaling pathways leading to apoptosis and tissue injury as well as reduction of cell function, which finally promote progression of cardiovascular disease. In regard to the design of new drugs, the understanding of the role of oxidative stress and the mechanism of redox signaling in initial steps of cardiovascular disease may open new avenues to develop new and more effective therapies against heart and blood vessel disorders. This chapter describes the impact of oxidative stress in the pathogenesis of hypertension, and explains the development of atherosclerosis and the effect of oxidative stress in this process. It also describes the effect of oxidative stress on progression of ischemia/reperfusion injury. The chapter reviews the pathogenesis of cardiac arrhythmia and the impact of oxidative stress on development of this disease." @default.
- W2258124546 created "2016-06-24" @default.
- W2258124546 creator A5021490438 @default.
- W2258124546 creator A5080937883 @default.
- W2258124546 date "2016-01-29" @default.
- W2258124546 modified "2023-10-17" @default.
- W2258124546 title "Reactive oxygen species, oxidative stress, and cardiovascular diseases" @default.
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