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- W2259195267 abstract "Intravenously injected NaSCN (20 to 100 mg/kg) markedly increased transmission through spinal monosynaptic and polysynaptic pathways. Such excitatory effects were seen in the neuronally isolated spinal cord. The excitatory effect of SCN was delayed in onset and was of short duration; median time to peak effect was 20 to 25 minutes, and the effect usually lasted less than 2 hours. In addition to its effect on excitation, SCN also enhanced synaptic recovery in the monosynaptic pathway. Intravenous injections of NaSCN produced immediate, but transient, increases in blood pressure (generally 30 to 60 mm Hg). Central excitatory effects of SCN were not causally related to the pressor effect of the drug. Intracellular recordings from motoneurons revealed marked effects of SCN. Following SCN motoneurons could be discharged by orthodromic impulses which were previously ineffective. Conduction block which prevented antidromic invasion of the motoneuron was also relieved by SCN. A striking feature of the intracellular recordings made after SCN was the increase in amplitude and duration of the negative after- potential associated with the motoneuron spike. In some instances repetitive motoneuron discharges were seen after SCN." @default.
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- W2259195267 date "1961-07-01" @default.
- W2259195267 modified "2023-09-23" @default.
- W2259195267 title "Excitatory effects of thiocyanate on spinal cord." @default.
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