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- W2261220138 abstract "Il existe une activation précoce de la coagulation chez les patients présentant un traumatisme crânien grave. Cette activation est d’origine intracérébrale, en relation probable avec la libération du facteur tissulaire. La présence de troubles de l’hémostase est un critère de gravité chez ces patients. L’activation de la coagulation peut conduire à une coagulation intravasculaire disséminée et à une fibrinolyse. La coagulation intravasculaire peut entraîner l’apparition de lésions ischémiques cérébrales par thrombose de la microcirculation. Cette revue s’intéresse à la physiopathologie de ces troubles de l’hémostase et à leurs conséquences thérapeutiques éventuelles.Early activation of coagulation is common after traumatic brain injury. Its origin is probably mainly intracerebral, due to tissue factor release from the injured brain. Abnormalities in blood coagulation tests are associated with poor neurological prognosis. Coagulation activation may induce disseminated intravascular coagulation and fibrinolysis. Disseminated intravascular coagulation is linked to brain ischemia caused by intravascular microthrombosis. This review will focus on pathophysiology of coagulation disorders after traumatic brain injury, and on their implications for therapeutic approaches." @default.
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- W2261220138 date "1968-12-01" @default.
- W2261220138 modified "2023-09-27" @default.
- W2261220138 title "Systemic Hemostatic Failure in the Severely Injured Patient" @default.
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- W2261220138 doi "https://doi.org/10.1016/s0039-6109(16)38684-4" @default.
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