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• W2264075160 abstract "Neutrophils infiltration/activation following wound induction marks the early inflammatory response in wound repair. However, the role of the infiltrated/activated neutrophils in tissue regeneration/proliferation during wound repair is not well understood. Here, we report that infiltrated/activated neutrophils at wound site release pyruvate kinase M2 (PKM2) by its secretive mechanisms during early stages of wound repair. The released extracellular PKM2 facilitates early wound healing by promoting angiogenesis at wound site. Our studies reveal a new and important molecular linker between the early inflammatory response and proliferation phase in tissue repair process." @default.
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• W2264075160 date "2016-03-01" @default.
• W2264075160 modified "2023-09-30" @default.
• W2264075160 title "PKM2 released by neutrophils at wound site facilitates early wound healing by promoting angiogenesis" @default.
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• W2264075160 doi "https://doi.org/10.1111/wrr.12411" @default.
• W2264075160 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/5873584" @default.
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