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• W2275017067 startingPage "469" @default.
• W2275017067 abstract "Infection in the immature low-birth-weight creased activation of the classical complement (LBW) infant is considered a major factor de- pathway.4 In addition low levels of C3 and factor termining morbidity and mortality. It has become B decrease amplification of the alternative pathincreasingly clear that the signs and symptoms of way.5 Diminished concentrations of the compomultiple organ failure associated with sepsis can nents of complement in the neonate may also result from infection as well as severe malnutri- lead to reduced deposition of opsonic C3 fragtion per se. Current nutritional support appears ments on the bacterial surface and to decreased to reduce infectious morbidity and mortality by generation of chemotactic factor C5a.4*” Plasma preventing deficiency of specific nutrients (eg, fibronectin levels in the newborn are one third zinc, retinol) and by securing adequate protein- to one half of those found in healthy adult subenergy supply. The concept of nutrients as de- jects. The rate of fibronectin synthesis is diminterminants of cell function, cytokine release, and ished in the LBW neonate; fibronectin half-life as regulators of the inflammatory and reparative and gestational age are inversely related.‘*s The processes has evolved over the past decades. It presence of common disease processes such as is in this setting that nutrients are proposed as respiratory distress syndrome, perinatal asphyxia, key modulators of the host defense response in bacterial sepsis, intrauterine growth retardation, LBW infants. or postnatal malnutrition determine further depression of the plasma fibronectin concentra" @default.
• W2275017067 created "2016-06-24" @default.
• W2275017067 creator A5021807554 @default.
• W2275017067 creator A5049530781 @default.
• W2275017067 date "1991-12-01" @default.
• W2275017067 modified "2023-10-12" @default.
• W2275017067 title "Nutrition and neonatal immune function." @default.
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• W2275017067 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/1803524" @default.
• W2275017067 hasPublicationYear "1991" @default.
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