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- W2275269915 endingPage "707" @default.
- W2275269915 startingPage "705" @default.
- W2275269915 abstract "脑肾素血管紧张素系统（RAS）在高血压的发展中起着重要的作用,主要是通过调节自主神经活性和血管加压素的释放来发挥作用。然而,脑是如何合成血管紧张素Ⅱ,长期以来存在争议,主要归究于中枢神经系统的低肾素活性。本文对新发现的RAS组分：脑肾素（原）受体,在调节血管紧张素Ⅱ形成和高血压的发展中的研究进展进行综述。" @default.
- W2275269915 created "2016-06-24" @default.
- W2275269915 creator A5000169719 @default.
- W2275269915 creator A5077883644 @default.
- W2275269915 date "2014-01-01" @default.
- W2275269915 modified "2023-09-23" @default.
- W2275269915 title "脑肾素（原）受体与神经源性高血压" @default.
- W2275269915 hasPublicationYear "2014" @default.
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