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• W2278318908 abstract "In the early fifties, Jacob Fine and his group [1] were the first to report, in the dog, that transmural migration of Escherichia coli from the gut occurred after prolonged chemical irritation of the peritoneal cavity. In 1966, Evans and Darin [2] demonstrated that a 90% distal enterectomy protected dogs from endotoxic shock; survival was improved from 13% in control animals to 52% in enterectomy treated animals. Accordingly, the authors suggested this was related to the inhibition of the translocation of bacteria, or bacterial-derived products, from the gut. Based on a growing body of evidence, Meakins and Marshall [3] later proposed in a review article that the gastrointestinal tract has a role in the pathogenesis of multiple organ failure (MOF), and may act as a ‘motor’ of this syndrome. They presented gut mucosal dysoxia as a consequence of an inadequate splanchnic oxygen delivery (DO2) to oxygen uptake (VO2) during shock states and as a cause for loss in the gut mucosal barrier, with bacterial or endotoxin translocation leading, ultimately, to remote organ injury and MOF (Fig. 1)." @default.
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• W2278318908 date "1999-01-01" @default.
• W2278318908 modified "2023-09-24" @default.
• W2278318908 title "The Role of the Gut in Multiple Organ Failure" @default.
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• W2278318908 doi "https://doi.org/10.1007/978-3-662-13453-5_46" @default.
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