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• W2282363463 abstract "This chapter provides a brief overview of the pathogenic strategies of some bacteria that infect the mucosal surface of the intestinal tract. It focuses on two model systems for the study of bacterial pathogens that cause disease by colonizing (enteropathogenic Escherichia coli [EPEC]) or penetrating (Salmonella species) the intestinal epithelium. Mucosal surfaces have many physiological defenses against pathogenic bacteria. These include entrapment in a thick blanket of mucus and clearance by peristalsis in the gut or ciliary movement in the airways. Adhesins on the bacterial surface provide specificity for interaction with target host cells. For example, M cells of the intestinal epithelium have cell surface glycosylation patterns that vary between species and tissue location. Adhesion is often a prerequisite for penetration of the mucosal surface, though different pathogens penetrate this barrier by different means and with different ends. EPEC provides a suitable model for understanding A/E pathogens and has largely been studied in vitro by infection of epithelial tissue cell cultures. Salmonella species infect a broad range of animals and can cause different diseases in different hosts. For example Salmonella enterica serovar Typhi causes typhoid fever in humans, which can be fatal. Recent progress has revealed the mechanisms by which the translocated effectors of SPI-1 mediate invasion by Salmonella serovar Typhimurium. Pathogenic bacteria have evolved different strategies to initiate infection at mucosal surfaces." @default.
• W2282363463 created "2016-06-24" @default.
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• W2282363463 date "2000-04-25" @default.
• W2282363463 modified "2023-10-17" @default.
• W2282363463 title "Bacterial Adherence, Colonization, and Invasion of Mucosal Surfaces" @default.
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• W2282363463 doi "https://doi.org/10.1128/9781555818111.ch1" @default.
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