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- W2287142148 abstract "Impaired pulmonary function continues to be a well-described but not a clinically well-appreciated complication of acute head injury. The process initially received wide attention beginning in the 1960s. Simmons et al. [1] reported that 85% of their series of combat casualties from Vietnam who died with a severe isolated head injury demonstrated significant pulmonary pathology including alveolar edema, hemorrhage, and congestion that was not the result of direct lung injury such as chest trauma. The findings were especially prominent in patients dying within the first 24 h as a result of severe neurologic impairment. Fulminant pulmonary edema on admission was evident in some of these patients. The term neurogenic pulmonary edema (NPE) was used to describe this very dramatic process and considerable research on NPE was initiated particularly in the 1970s [2, 3]. However, fulminant early lung edema was not common in a more typical cross section of civilians with head injuries, except in young patients with massive and usually rapidly fatal brain damage [4]. Most NPE series have come from autopsy studies [1]. The true clinical relevance of NPE in nonfatal injuries remains poorly defined, since its incidence in the less massively injured population appears to be extremely low." @default.
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- W2287142148 date "1991-01-01" @default.
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- W2287142148 title "Lung Dysfunction After Head Injury" @default.
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- W2287142148 doi "https://doi.org/10.1007/978-3-642-84423-2_54" @default.
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