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• W2288654312 startingPage "458" @default.
• W2288654312 abstract "The two primary sites of herbicide action in photosynthetic electron transport are the inhibition of photosystem II (PS II) electron transport and diversion of electron flow through photosystem I (PS I). PS II electron transport inhibitors bind to the D1 protein of the PS II reaction center, thus blocking electron transfer to plastoquinone. Inhibition of PS II electron transport prevents the conversion of absorbed light energy into electrochemical energy and results in the production of triplet chlorophyll and singlet oxygen which induce the peroxidation of membrane lipids. PS I electron acceptors probably accept electrons from the iron-sulfur protein, F a /F b . The free radical form of the herbicide leads to the production of hydroxyl radicals which cause the peroxidation of lipids. Herbicide-induced lipid peroxidation destroys membrane integrity, leading to cellular disorganization and phototoxicity." @default.
• W2288654312 created "2016-06-24" @default.
• W2288654312 creator A5036455715 @default.
• W2288654312 creator A5073774658 @default.
• W2288654312 date "1991-09-01" @default.
• W2288654312 modified "2023-10-12" @default.
• W2288654312 title "Interactions of Herbicides with Photosynthetic Electron Transport" @default.
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• W2288654312 doi "https://doi.org/10.1017/s0043174500073227" @default.
• W2288654312 hasPublicationYear "1991" @default.
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