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- W2290626972 endingPage "215" @default.
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- W2290626972 abstract "Genetic variants in the Apolipoprotein L1 (APOL1) gene cause high rates of kidney disease in African Americans. These variants, found only in individuals with recent African ancestry, confer enhanced innate immunity against African trypanosomes. Although they are among the most powerful disease-causing common variants discovered to date, we are just beginning to understand how they promote kidney injury. Since APOL1 is present in only a few primate species, much of our current knowledge has come from natural experiments in humans and in vitro studies while awaiting the development of transgenic animal models. Understanding more about the function of ApoL1 and how the high-risk variants behave differently from other ApoL1 molecules is a high priority in kidney disease research." @default.
- W2290626972 created "2016-06-24" @default.
- W2290626972 creator A5020267280 @default.
- W2290626972 creator A5047201622 @default.
- W2290626972 date "2016-04-01" @default.
- W2290626972 modified "2023-10-06" @default.
- W2290626972 title "Apolipoprotein L1 and Kidney Disease in African Americans" @default.
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- W2290626972 doi "https://doi.org/10.1016/j.tem.2016.02.002" @default.
- W2290626972 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/4811340" @default.
- W2290626972 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/26947522" @default.
- W2290626972 hasPublicationYear "2016" @default.
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