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• W2291947617 abstract "The role of genetic factors in essential hypertension is supported by studies of subjects with known genetic relationships (family studies) (41–43) monozygotic twins (25, 26, 37, 38), natural vs adopted children (18) and experimental models of spontaneous or “genetic” hypertension in rats (6, 24, 45). The demonstration that genetic factors are involved in this disease has stimulated many investigators to look for possible biochemical-functional alterations that could be considered as the direct phenotypic expression of genetic abnormalities underlying hypertension. Thus many studies have been performed on human hypertension to investigate alterations of transmembrane ionic transport, intracellular ion content and molecular characteristics of cellular membranes. However, these studies (1, 18, 21, 22, 33, 61) have not resulted in a consistent picture characteristic of the hypertensive patient. This is not surprising considering that essential hypertension is a polygenic disease, phenotypically expressed in different ways depending on the interaction between many environmental factors and a polygenic substrate. When approaching the problem of the etiology of human essential hypertension, it is therefore important to study the same patients with different techniques of physiology, biochemistry, molecular biology and population genetics in order to clarify the sequence of events from genetic alteration to the cellular and organ alteration responsible for the disease in the individual patient or family." @default.
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• W2291947617 date "1989-01-01" @default.
• W2291947617 modified "2023-09-26" @default.
• W2291947617 title "The genetics of essential hypertension" @default.
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• W2291947617 doi "https://doi.org/10.1007/978-3-642-72417-6_7" @default.
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