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- W2292210286 abstract "Anemia is the primary clinical manifestation of malarial infections and is responsible for the substantial rate of morbidity. The pathophysiology discussed till now catalogued several causes for malarial anemia among which ineffective erythropoiesis being remarkable one occurs silently in the bone marrow. A systematic literature search was performed and summarized information on erythropoietic response upon malaria infection and the factors responsible for the same. This review summarizes the clinical and experimental studies on patients, mouse models, and in vitro cell cultures reporting erythropoietic changes upon malaria infection as well as factors accountable for the same. Inadequate erythropoietic response during malaria infection may be the collective effect of various mediators generated by host immune response as well as parasite metabolites. The interplay between various modulators causing the pathophysiology needs to be explored further. Globin gene expression profiling upon malaria infection should also be looked into as abnormal production of globin chains could be a possible contributor to ineffective erythropoiesis." @default.
- W2292210286 created "2016-06-24" @default.
- W2292210286 creator A5024269433 @default.
- W2292210286 creator A5031782697 @default.
- W2292210286 date "2016-01-01" @default.
- W2292210286 modified "2023-10-18" @default.
- W2292210286 title "Erythropoiesis in Malaria Infections and Factors Modifying the Erythropoietic Response" @default.
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- W2292210286 doi "https://doi.org/10.1155/2016/9310905" @default.
- W2292210286 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/4789361" @default.
- W2292210286 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/27034825" @default.
- W2292210286 hasPublicationYear "2016" @default.
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