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- W2292519795 endingPage "828" @default.
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- W2292519795 abstract "Endothelial cells transduce the frictional force from blood flow (fluid shear stress) into biochemical signals that regulate gene expression and cell behavior via specialized mechanisms and pathways. These pathways shape the vascular system during development and during postnatal and adult life to optimize flow to tissues. The same pathways also contribute to atherosclerosis and vascular malformations. This Review covers recent advances in basic mechanisms of flow signaling and the involvement of these mechanisms in vascular physiology, remodeling, and these diseases. We propose that flow sensing pathways that govern normal morphogenesis can contribute to disease under pathological conditions or can be altered to induce disease. Viewing atherosclerosis and vascular malformations as instances of pathological morphogenesis provides a unifying perspective that may aid in developing new therapies." @default.
- W2292519795 created "2016-06-24" @default.
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- W2292519795 creator A5056681968 @default.
- W2292519795 creator A5086030901 @default.
- W2292519795 creator A5087900306 @default.
- W2292519795 date "2016-03-01" @default.
- W2292519795 modified "2023-10-14" @default.
- W2292519795 title "Endothelial fluid shear stress sensing in vascular health and disease" @default.
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