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- W2299446503 abstract "Renal and lung epithelial cells are exposed to some significant concentrations of H 2 O 2 . In urine it may reach 100 μ M, while in the epithelial lining fluid in the lung it is estimated to be in micromolar to tens-micromolar range. Hydrogen peroxide has a stimulatory action on the epithelial sodium channel (ENaC) single-channel activity. It also increases stability of the channel at the membrane and slows down the transcription of the ENaC subunits. The expression and the activity of the channel may be inhibited in some other, likely higher, oxidative states of the cell. This review discusses the role and the origin of H 2 O 2 in the lung and kidney. Concentration-dependent effects of hydrogen peroxide on ENaC and the mechanisms of its action have been summarized. This review also describes outlooks for future investigations linking oxidative stress, epithelial sodium transport, and lung and kidney function." @default.
- W2299446503 created "2016-06-24" @default.
- W2299446503 creator A5030792958 @default.
- W2299446503 creator A5044380183 @default.
- W2299446503 creator A5054910178 @default.
- W2299446503 date "2016-01-01" @default.
- W2299446503 modified "2023-10-17" @default.
- W2299446503 title "Hydrogen Peroxide and Sodium Transport in the Lung and Kidney" @default.
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- W2299446503 doi "https://doi.org/10.1155/2016/9512807" @default.
- W2299446503 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/4814630" @default.
- W2299446503 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/27073804" @default.
- W2299446503 hasPublicationYear "2016" @default.
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