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- W230188108 abstract "Exposure to a traumatic event is required for the diagnosis of posttraumatic stress disorder (PTSD). The symptoms of PTSD are believed to reflect stress-induced changes in neurobiological systems, an inadequate adaptation of neurobiological systems to exposure to severe stressors, or both. More recently, there have been attempts, such as via brain mapping, to identify neurobiological changes to the specific, altered neuroanatomical features that constitute PTSD. Additionally, there have been efforts to understand whether specific neurobiological changes in PTSD reflect preexisting susceptibility factors rather than consequences of trauma exposure or correlates of PTSD. Altercations in genome and downstream changes in gene expression and protein functions, genetic variability, sex differences, and developmental exposures to stress and chemical toxins influence neurobiological systems and moderate PTSD risk. Currently, the advanced genomic and proteomic high-throughput methodologies that are applied effectively in understanding the molecular signatures of many neurological disorders can also be distinctively applied to PTSD also. On the basis of these findings, important hypotheses for developing novel strategies to identify subjects at risk, promote resilience, and devise targets for the prevention or treatment of PTSD can be derived." @default.
- W230188108 created "2016-06-24" @default.
- W230188108 creator A5033337304 @default.
- W230188108 creator A5052747154 @default.
- W230188108 creator A5089712382 @default.
- W230188108 date "2015-01-01" @default.
- W230188108 modified "2023-09-23" @default.
- W230188108 title "Genomics and Proteomics in Brain Complexity in Relation to Chemically Induced PTSD" @default.
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