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• W2302223954 endingPage "e1162369" @default.
• W2302223954 startingPage "e1162369" @default.
• W2302223954 abstract "The majority of the vascular flowering plants form symbiotic associations with fungi from the phylum Glomeromycota through which both partners gain access to nutrients, either mineral nutrients in the case of the plant, or carbon, in the case of the fungus.Citation1 The association develops in the roots and requires substantial remodeling of the root cortical cells where branched fungal hyphae, called arbuscules, are housed in a new membrane-bound apoplastic compartment.Citation2 Nutrient exchange between the symbionts occurs over this interface and its development and maintenance is critical for symbiosis. Previously, we showed that DELLA proteins, which are well known as repressors of gibberellic acid signaling, also regulate development of AM symbiosis and are necessary to enable arbuscule development.Citation3 Furthermore, constitutive overexpression of a dominant DELLA protein (della1-Δ18) is sufficient to induce transcripts of several AM symbiosis-induced genes, even in the absence of the fungal symbiont.Citation4 Here we further extend this approach and identify AM symbiosis genes that respond transcriptionally to constitutive expression of a dominant DELLA protein and also genes that do respond to this treatment. Additionally, we demonstrate that DELLAs interact with REQUIRED FOR ARBUSCULE DEVELOPMENT 1 (RAD1) which further extends our knowledge of GRAS factor complexes that have the potential to regulate gene expression during AM symbiosis." @default.
• W2302223954 created "2016-06-24" @default.
• W2302223954 creator A5005319312 @default.
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• W2302223954 creator A5063600156 @default.
• W2302223954 date "2016-03-16" @default.
• W2302223954 modified "2023-10-18" @default.
• W2302223954 title "DELLA proteins regulate expression of a subset of AM symbiosis-induced genes in<i>Medicago truncatula</i>" @default.
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• W2302223954 doi "https://doi.org/10.1080/15592324.2016.1162369" @default.
• W2302223954 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/4883957" @default.
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• W2302223954 hasPublicationYear "2016" @default.
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