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- W2304366241 endingPage "26" @default.
- W2304366241 startingPage "15" @default.
- W2304366241 abstract "Although the genetic profile of pancreatic cancer is emerging as a result of much research, the role of specific genetic alterations that initiate tumorigenesis and produce its cardinal clinical features of locally aggressive growth, metastasis, and chemotherapy resistance remains unresolved. Recently, a number of studies have shown that the inhibition of constitutive NF-kappaB activation, one of the frequent molecular alterations in pancreatic cancer, inhibits tumorigenesis and metastasis. It also sensitizes pancreatic cancer cell lines to anticancer agent-induced apoptosis. Therefore because of the crucial role of NF-kappaB in pancreatic cancer, it is a potential target for developing novel therapeutic strategies for the disease. In vivo and in vitro models that mimic the tumorigenic phenotypes in the appropriate histological and molecular concert would be very useful for confirming the suspected role of the pancreatic cancer signature genetic lesions and better understanding the molecular basis of this disease." @default.
- W2304366241 created "2016-06-24" @default.
- W2304366241 creator A5031736405 @default.
- W2304366241 creator A5040956378 @default.
- W2304366241 creator A5045763436 @default.
- W2304366241 creator A5068301962 @default.
- W2304366241 creator A5076270097 @default.
- W2304366241 date "2003-01-01" @default.
- W2304366241 modified "2023-10-16" @default.
- W2304366241 title "NF-κB in Pancreatic Cancer" @default.
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