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- W2310282931 startingPage "R676" @default.
- W2310282931 abstract "Systemic injection of the fructose analogue 2,5-anhydro-D-mannitol (2,5-AM) elicits a feeding response and induces c-fos activity in the parabrachial nuclei (PBN). We used bilateral ibotenic acid lesions of PBN to determine whether the activation inferred from c-fos activity was causally related to the feeding response. The relationship between the PBN lesion and feeding behavior was also examined with the glucose analogue 2-deoxy-D-glucose (2-DG). The PBN lesions interfered with the feeding response to 2,5-AM but spared the feeding response to 2-DG. Rats were also tested in a conditioned taste-aversion paradigm. Differences were observed in the relationship between lesion extent and behavioral deficit for feeding responses to 2,5-AM and taste-guided intake after taste-aversion conditioning. These data provide the first demonstration that central lesions can disrupt feeding responses to peripherally acting 2,5-AM. The results suggest that the neural substrate for this response differs from that mediating taste-aversion conditioning and from that involved in the feeding response to 2-DG." @default.
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- W2310282931 date "1995-03-01" @default.
- W2310282931 modified "2023-09-25" @default.
- W2310282931 title "Parabrachial nucleus lesions impair feeding response elicited by 2,5-anhydro-D-mannitol" @default.
- W2310282931 doi "https://doi.org/10.1152/ajpregu.1995.268.3.r676" @default.
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