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- W2314359946 endingPage "G476" @default.
- W2314359946 startingPage "G460" @default.
- W2314359946 abstract "Inflammatory bowel disease (IBD) is a chronic inflammatory disorder with a complex pathogenesis. Diarrhea is a highly prevalent and often debilitating symptom of IBD patients that results, at least in part, from an intestinal hydroelectrolytic imbalance. Evidence suggests that reduced electrolyte absorption is more relevant than increased secretion to this disequilibrium. This systematic review analyses and integrates the current evidence on the roles of epithelial Na + -K + -ATPase (NKA), Na + /H + exchangers (NHEs), epithelial Na + channels (ENaC), and K + channels (KC) in IBD-associated diarrhea. NKA is the key driving force of the transepithelial ionic transport and its activity is decreased in IBD. In addition, the downregulation of apical NHE and ENaC and the upregulation of apical large-conductance KC all contribute to the IBD-associated diarrhea by lowering sodium absorption and/or increasing potassium secretion." @default.
- W2314359946 created "2016-06-24" @default.
- W2314359946 creator A5045428922 @default.
- W2314359946 creator A5052535018 @default.
- W2314359946 creator A5064221651 @default.
- W2314359946 creator A5078712323 @default.
- W2314359946 date "2016-04-01" @default.
- W2314359946 modified "2023-10-11" @default.
- W2314359946 title "Role of epithelial ion transports in inflammatory bowel disease" @default.
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