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- W2315670094 abstract "Term and preterm neonates experience quantitative and qualitative neutrophil deficiencies resulting in part from decreased production of granulocyte colony-stimulating factor (G-CSF). In adults, G-CSF improves neutrophil function by up-regulating adhesion molecules.To evaluate the effects of G-CSF on neonatal neutrophil adhesive phenotypes, cord blood samples were incubated with G-CSF or phosphate-buffered saline and stimulated with N-formyl-methionyl-leucyl-phenylalanine (FMLP), and adhesion molecules were evaluated by flow cytometry.In term and preterm neutrophils, G-CSF incubation increased beta2-integrin expression significantly compared with baseline and to a greater extent than observed in adult neutrophils. With FMLP stimulation, beta2-integrin expression increased even more in the G-CSF group. L-selectin expression decreased after G-CSF incubation and decreased even more with FMLP stimulation in the G-CSF group compared with the phosphate-buffered saline group in term and preterm samples, but not in adult samples.The data show that G-CSF increases expression of beta2-integrin and decreases expression of L-selectin on unstimulated and stimulated term and preterm neonatal neutrophils in vitro. Further study is required to determine whether G-CSF improves neonatal neutrophil function." @default.
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- W2315670094 date "2001-11-01" @default.
- W2315670094 modified "2023-09-27" @default.
- W2315670094 title "Effects of Granulocyte Colony-Stimulating Factor on Neutrophil Adhesive Molecules in Neonates" @default.
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- W2315670094 doi "https://doi.org/10.1097/00043426-200111000-00008" @default.
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