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- W2317366097 abstract "lewisi infections. Although there were some differences, the hyperimmune antiserum showed several lines of identity with antisera collected at various times during a typical primary infection. Antisera collected 21 and 28 days postinfection shared two precipitin lines with the hyperimmune rat antisera, and antisera collected from 49 to 250 days postinfection produced from three to four common precipitin lines. Why these antibodies are not usually detected in the serum of rats with a typical primary infection is unknown. The presence of antibodies in the sera of infected rats has been suggested by Long and Dusanic (1978, loc. cit.). If antibodies are present, then exoantigens might be released as antigen-antibody complexes in the untreated rats. The ability of hyperimmune antisera to precipitate exoantigens from the immunocompetent rats may be the result of increased avidity or the presence of antibody i ections. Although there were some to the antigen-antibody complexes. In the immunosuppressed host, these exoantigens might be released as free antigens, which would account for their increased reactivity. Ferrante and Jenkin (1977, Austral. J. Exp. Biol. Med. Sci. 55: 275-289) detected nonopsonic, noncomplement fixing, blocking antibodies on the surface of T. lewisi and suggested that these immunoglobulins played a role in protecting the parasites from trypanocidal antibodies. Giannini and D'Alesandro (1979, Exp. Parasitol. 47: 342-355) suggested that the bound immunoglobulin is the reproduction inhibiting antibody ablastin. The exact nature and function of antibodies bound to T. lewisi remains to be determined. This work was supported in part by Grant AI-11369 from the U.S. National Institutes of Health and National Science Foundation Grants PCM 76-11922 and PCM 78-23939. the antigen-antibody complexes. In the im-" @default.
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- W2317366097 date "1983-02-01" @default.
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- W2317366097 title "Babesia bovis: attachment of infected erythrocytes to heparin-sepharose columns." @default.
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