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- W2319665226 abstract "Background and Aims: Hepatitis B virus (HBV) infection can result in numerous different clinical outcomes. Complex interaction of viral, genetic and environmental factors seems to be responsible for both susceptibility to HBV persistence and the course of infection. To assess the possible role of cytokine (IL-8) in liver damage of hepatitis B Methods: A total of 100 patients with HBV infection and 50 healthy blood donors were studied. Samples were collected from the patients attending the outpatient department and admitted in the wards of Medicine of Lok Nayak Jay Prakash Hospital (MAMC), New Delhi. Serum IL-8 levels were measured by using commercially available ELISA kits as per the manufacturer instruction [Ray Biotech, Norcross]. There were 50 cases of cirrhosis, 50 cases of chronic hepatitis and 50 healthy blood donors. The serological markers of HAV, HCV and HEV were negative, and HBsAg and other markers of HBV were positive in all the patients. Results: IL-8 in all types of clinical hepatitis B were significantly higher than those in healthy blood donors (P <0.05). IL-8 were positively correlated with serum bilirubin (P <0.05).IL- 8 is elevated more in liver cirrhosis with positive HBeAg in comparison to control. The IL-8 levels calculated were Cirrhosis (41.66±50.47), Chronic Hepatitis B (31.94±47.88) and Healthy (10.31±7.42) Pico grams/ml. Conclusion: Significant difference was observed in mean IL-8 levels in patients of cirrhosis as compared to controls. But the difference was not significant for IL-8 in chronic hepatitis B as compared to healthy controls. IL-8 levels are increased predominantly when liver diseases become active." @default.
- W2319665226 created "2016-06-24" @default.
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- W2319665226 date "2013-03-01" @default.
- W2319665226 modified "2023-10-16" @default.
- W2319665226 title "Changes in levels of interleukin-8 in the serum of patients with hepatitis B virus infection in north indian population" @default.
- W2319665226 doi "https://doi.org/10.1016/j.jceh.2013.03.135" @default.
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